Arterial Thrombosis and its pathogenesis are dynamic and complex. Dissimilar to venous thrombi, the category of thrombosis usually form under high blood flow conditions, and are chiefly composed of aggregates of platelet, which give them the looks of white clots.
Huge evidence suggests the Arterial Thrombosis to be originated as a condition of an injured plaque of atherosclerosis. Their formation result in the release of platelet aggregation, prothrombotic material, and adhesion of platelets to the wall. The platelet plaque at the initial level is then calmed by fibrin production on the activation of the coagulation cascade.
The acquired conditions of predisposing and inherited gene polymorphisms can manipulate both the size and composition of an Arterial Thrombosis. Additional research is needed to clarify the functions of cellular elements and blood coagulation protein, which are serious to the Arterial pathogenesis. Continue reading “Arterial Thrombosis”